微信打中文自动翻译成英文
微信输入文字自动转换为英文，可以在微信聊天框发送三句相同的英文，页面弹出边写边译点击，即可文字自动转换为英文，以下是具体操作步骤：1.发送三条相同英文进入一个微信好友聊天对话框，连续向此微信好友发送三条相同的英文。2.点击边写边译输入框弹出小标题，长按输入框可以开启，边写边译，根据提示长按输入框，弹出选项选择边写边译。3.点击使用在输入框输入一段中文，文字上方将自动转换出英文，点击使用即可。微信（WeChat）是腾讯公司于2011年1月21日推出的一个为智能终端提供即时通讯服务的免费应用程序 。微信支持跨通信运营商、跨操作系统平台通过网络快速发送免费（需消耗少量网络流量）语音短信、视频、图片和文字，同时，也可以使用通过共享流媒体内容的资料和基于位置的社交插件“摇一摇”、“朋友圈”、”公众平台“、”语音记事本“等服务插件。截止到2016年第二季度，微信已经覆盖中国94%以上的智能手机，月活跃用户达到8.06亿， 用户覆盖200多个国家和地区、超过20种语言 。此外，各品牌的微信公众账号总数已经超过800万个，移动应用对接数量超过85000个，广告收入增至36.79亿人民币 ，微信支付用户则达到了4亿左右 。
微信怎么翻译成英文发出去
微信里输入法用搜狗输入法则可直接将中文转换成英文。1、以手机版微信为例，首先打开微信后，在对话框输入你要发送的内容。2、然后全选要发送的内容，下面就会出现多种语言供选择，选择里面的英文格式。3、这时所输入的中文就变成英文的了，直接点击右下角的发送则可。微信的介绍：是中国腾讯公司于2011年1月21日推出的一款支持Android以及iOS等移动操作系统的即时通信软件，其面对智能手机用户。用户可以透过客户端与好友分享文字、图片以及贴图，并支持分组聊天和语音、视讯对讲功能、广播（一对多）消息、照片/视讯共享、位置共享、消息交流联系、微信支付、理财通，游戏等服务。微信支持多种语言，以及手机数据网络。用户可拍摄照片或视频发送至“朋友圈”。用户可在联系人列表中选择联系人，使用云端服务将数据备份和恢复，以保护用户通讯录数据。微信中还有订阅号、服务号、企业号等功能，可以供用户订阅他们喜欢的公众号，也提供了一个良好的自媒体平台，每个人都可以申请个人订阅号发布个人的文章等，用户可以透过订阅或者搜索获取微信公众号的文章。
翻译 (英译中)
Confirmed received and thank you. 你送来的 (文件 / 货品) 已收妥. 谢谢 Unless you just want to send originals by courier
scanned copies are fine. 我们只需要 (文件的) 电子扫描版. 如果你认为有必要
也可以用速递送原件来. (原文是很简洁
很 INFORMAL 的商业英文
看来用在EMAIL 里)
(已帮你英译中): Confirmed received and thank you. 证实被确认多谢. Unless you just want to send originals by courier
除非你想传送原本的急件
scanned copies are fine. 详看副本是好的. [希 望帮到你] 图片参考：.yimg/iugc/rte/ *** iley_39
Confirmed received and thank you. Unless you just want to send originals by courier
scanned copies are fine. 兹确认妥收及谨此致谢
除非你认为要以速递交来原稿
否则电脑之复件已可被接纳. 2008-06-12 08:35:04 补充： 电脑扫描之复件 ( 会较详细 ) 2008-06-12 10:17:44 补充： fine = good enough
证实接受和谢谢。 除非您想要由传讯者送原物，被扫描的拷贝优良是。
确认收到，谢谢。除非你只是想寄原件通过快递公司，扫描副本的罚款。 Confirmed received and thank you. Unless you just want to send originals by courier
scanned copies are fine.
上面个位唔系翻译得好对窝．． 证实收到［你份野］喇，多谢晒！ 除非你只想将原装版本速递［过黎］， 你个d scan左入电脑既文件就OK喇。 2008-06-11 12:13:58 补充： 我指001个位
Confirmed received and thank you. 证实被确认多谢. Unless you just want to send originals by courier
除非你想传送原本的急件
scanned copies are fine. 详看副本是好的. 2008-06-11 13:51:58 补充： 003： 多谢提点㖞！！但我都唔会自信爆棚到乱话人错又D中文译成咁罗！！！
翻译 中译英
Male mice in the AOM/DSS model were randomly divided into 4 groups: Model group, Model+AAV-NC group, Model+AAV/NC+BBD group, and Model/OE-SNTB1+BBD group. After one week of adaptive feeding, mice were injected with 12.5mg/kg AOM. After one week, they were given 2.5% DSS to induce the construction of colitis-associated colon cancer (CAC) model. SNTB1 gene overexpression plasmid was packaged and recombinant virus particles were obtained. The colon tissue of the mice was infected with the virus particles. At the beginning of each round of DSS, mice in the Model+AAV-NC and Model+AAV/NC+BBD groups were injected with 100ul of empty adenovirus, while mice in the Model/OE-SNTB1+BBD group were injected with 100ul of SNTB1 overexpression adenovirus. After the first round of DSS feeding ended, mice were given BaBaoDan (250mg/kg) for long-term intervention. Tissue analysis was conducted on day 67 of the experiment. The general living conditions of the mice and their weight were observed. An in viv【摘要】翻译 中译英【提问】我这边已经机翻好了，需要您帮我看看语法什么的，检查和更改一下【提问】亲 发来看看呢【回答】发文字哦【回答】中文摘要目的：通过体内外实验，在体外水平研究八宝丹（BaBaoDan，BBD）治疗大肠癌细胞的作用机制，检验SNTB1是否是八宝丹发挥治疗大肠癌功能的潜在靶点；以及在体内水平研究检验八宝丹是否通过调控SNTB1的表达，影响p38MAPK信号通路活化，参与调控结肠炎相关性肠癌（CAC）的发生发展，从而为临床应用八宝丹治疗CAC奠定基础,也为结肠炎相关性结肠癌的治疗提供了基因治疗新的思路。方法：1.体外培养SNTB1、LOVO肠癌细胞，采用慢病毒感染和嘌呤霉素筛选构分别构建大肠癌稳转细胞株，将2株细胞分为HCT-116/NC组、HCT-116/OE组及LOVO/NC组、LOVO/OE组；采用MTT法、细胞克隆球形成实验、Transwell、Invasion实验分别检测转染后各组细胞的活力、增殖、迁移侵袭能力； qPCR法检测转染后各组SNTB1的表达情况；WB检测过表达组细胞中SNTB1的表达是否被上调以及MAPK12、PMAPK12表达能力。【提问】AbstractObjective: To investigate the mechanism of BaBaoDan (BBD) in the treatment of colorectal cancer cells in vitro and in vivo, and to test whether SNTB1 is a potential target for BaBaoDan to exert its therapeutic effect on colorectal cancer cells; "And in vivo level studies to test whether Babao Dan can regulate the expression of SNTB1, affect the activation of p38MAPK signaling pathway, and participate in the regulation of the occurrence and development of colitis associated colon cancer【提问】Abstract:The objective of this study is to investigate the mechanism of BaBaoDan (BBD) in treating colorectal cancer (CRC) cells in vitro and in vivo, and to verify whether SNTB1 is a potential target for the therapeutic effect of BBD on CRC. The study also aims to examine whether BBD can affect the activation of p38MAPK signaling pathway by regulating the expression of SNTB1 in vivo and participate in the regulation of the occurrence and development of colitis-associated colon cancer (CAC), thus laying a foundation for the clinical application of BBD in the treatment of CAC and providing a new approach to gene therapy for colitis-associated colon cancer.Methods:SNTB1 and LOVO CRC cells were cultured in vitro, and stable cell lines of CRC were constructed by lentivirus infection and puromycin screening. The two cell lines were divided into HCT-116/NC group, HCT-116/OE group, LOVO/NC group, and LOVO/OE group. MTT assay, cell clone sphere formation experiment, Transwell, and Invasion【回答】(CAC), thereby laying the foundation for the clinical application of BBD in the treatment of CAC and providing a new gene therapy approach for colitis-associated colon cancer.Methods: SNTB1 and LOVO colorectal cancer cells were cultured in vitro, and stable cell lines were constructed using lentivirus infection and puromycin screening. The cells were divided into HCT-116/NC, HCT-116/OE, LOVO/NC, and LOVO/OE groups. The cell viability, proliferation, migration, and invasion ability of each group after transfection were assessed using MTT assay, cell clone sphere formation experiment, Transwell, and Invasion assay. qPCR was used to detect the expression of SNTB1 in each group after transfection, and WB was used to detect whether the expression of SNTB1 was upregulated and the expression ability of MAPK12 and PMAPK12 in the overexpressed cells.【回答】【提问】【提问】亲 这个太长了吧，8毛钱很难做到啊[流泪]【回答】好的，感谢【提问】我慢慢来吧，一段一段发给您【回答】Objective: To investigate the mechanism of BaBaoDan (BBD) in the treatment of colorectal cancer cells in vitro and in vivo, and to verify whether SNTB1 is a potential target for BBD to exert its therapeutic effect on colorectal cancer cells. Additionally, the study aims to examine whether BBD can affect the activation of p38MAPK signaling pathway by regulating the expression of SNTB1 in vivo and participate in the regulation of the occurrence and development of colitis-associated colon cancer (CAC), thus laying a foundation for the clinical application of BBD in the treatment of CAC and providing a new gene therapy approach for colitis-associated colon cancer.Methods: SNTB1 and LOVO colorectal cancer cells were cultured in vitro, and stable cell lines were constructed using lentivirus infection and puromycin screening. The cells were divided into HCT-116/NC, HCT-116/OE, LOVO/NC, and LOVO/OE groups. The cell viability, proliferation, migration, and invasion ability of each group after【回答】中文摘要目的:通过体内外实验，在体外水平研究八宝丹(BaBaoDan，BBD)治疗大肠癌细胞的作用机制检验SNTB1是否是八宝丹发挥治疗大肠癌功能的潜在靶点;以及在体内水平研究检验八宝丹是否通过调控SNTB1的表达，影响p38MAPK信号通路活化，参与调控结肠炎相关性肠癌(CAC)的发生发展，从而为临床应用八宝丹治疗CAC奠定基础也为结肠炎相关性结肠癌的治疗提供了基因治疗新的思路。方法:1体外培养SNTB1、LOVO肠癌细胞，采用慢病毒感染和漂岭霉素筛选构分别构建大肠癌稳转细胞株，将2株细胞分为HCT-116/NC组、HCT-116/OE组及LOVO/NC组、LOVO/OE组;采用MTT法、细胞克隆坏成实觉Iranswellnvasion:染后各组细胞的活力、增殖、迁移侵袭能力:qPCR法检测转染后各组SNTB1的表达情况:WB检测过表达组细胞中SNTB1的表达是否被上调以及MAPK12PMAPK12表达能力。2.将构建成功后的大肠癌稳转细胞株采用MTT法、细胞克隆球形成实验、Transwell、invasionScratch实验分别检测转染后各组细胞的活力、增殖迁移侵袭、转移能力:qPCR法检测转染后各组SNTB1的表达情况WB检测过表达组细胞中SNTB的表达是否被上调以及MAPK12、PMAPK12表达能力，以检测八宝丹对SNTB1介导的大肠癌细胞模型的影响。【回答】这段的【回答】SNTB1 adenovirus was constructed, and male Balb/c mice were randomly divided into 5 groups: Model group, Model/OE-NC group, Model/OE group, Model/Sh-NC group, and Model/Sh group. The general living conditions of the mice, as well as their weight and fecal occult blood were observed. The virus infection of the colon tissue was observed using an in vivo imaging system. The virus infection effect was verified using frozen sections, and the expression of SNTB1, MAPK12, and PMAPK12 in colon tissue was detected using IHC. RT-PCR was used to detect the differences in SNTB1 mRNA expression in colon tissue among the groups of mice, and Western blot was used to detect the expression of SNTB1, MAPK12, and PMAPK12.【回答】3构建SNTB1腺相关病毒，将雄性Balb/c小鼠随机分成5组，Model组、ModeVOE-NC组Model/OE组、Model/Sh-NC组、Model/Sh组;观察小鼠一般生活状态和测量体重、粪便隐血情况，通过活体成像系统观察结肠组织病毒感染情况，冰冻切片验证病毒感染效果，通过IHC检测结肠组织SNTB1、MAPK12PMAPK12表达;利用RT-gPCR技术检测各组小鼠的结肠组织SNTB1mRNA表达差异，通过Westermn Blot检测SNTB1、MAPK12、PMAPK12表达。这段的【回答】Male mice in the AOM/DSS model were randomly divided into 4 groups: Model group, Model+AAV-NC group, Model+AAV/NC+BBD group, and Model/OE-SNTB1+BBD group. After one week of adaptive feeding, mice were injected with 12.5mg/kg AOM. After one week, they were given 2.5% DSS to induce the construction of colitis-associated colon cancer (CAC) model. SNTB1 gene overexpression plasmid was packaged and recombinant virus particles were obtained. The colon tissue of the mice was infected with the virus particles. At the beginning of each round of DSS, mice in the Model+AAV-NC and Model+AAV/NC+BBD groups were injected with 100ul of empty adenovirus, while mice in the Model/OE-SNTB1+BBD group were injected with 100ul of SNTB1 overexpression adenovirus. After the first round of DSS feeding ended, mice were given BaBaoDan (250mg/kg) for long-term intervention. Tissue analysis was conducted on day 67 of the experiment. The general living conditions of the mice and their weight were observed. An in viv【回答】4.将AOM/DSS模型小凰随机分成4组，分别是Model组、Model+AAV-NC组Model+AAV/NC+BBD组、Model/OE-SNTB1+BBD组，经适应性喂养一周后，对小鼠体内注射12.5mg/kg的氧化偶氮甲烷(AOM)，一周后，给予2.5%浓度的葡聚糖硫酸钠(DSS)，诱导构建结肠炎相关性结肠癌(CAC)模型。将构建的SNTB1基因过表达质粒包装、重组得到病毒颗粒，感染小鼠结肠组织部位，在每一轮DSS开始时，Model+AAV-NC组及Model+AAV/NC+BBD组小鼠注射100ul/只的空载腺病毒，Model/OE-SNTB1+BBD组小鼠注射100ul/只的SNTB1过表达腺病毒。在第一轮DSS喂养结束后odeltAAWNCRRDEB1+RRD6小鼠使用八宝丹(250mg/kg)长期干预:在实验第67天的时候进行取材分析。观察小鼠一般生活状态和检测体重，运用小动物活体成像系统观察结肠组织病毒感染情况;利用冰冻切片验证病毒感染效果，免疫组化检测SNTB1、MAPK12、PMAPK12阳性率评价长期使用八宝丹对脏器组织病理学的影响，HE染色检测结肠组织瘤体情况，RT-GPCR检测结肠组织SNTB1表达差异;Western Blot检测SNTB1蛋白、p-MAPK12/MAPK12通路的表达情况，以验证八宝丹是否是通过调控SNTB1基因影响结肠炎相关性结肠癌小鼠的发生发展。这段的【回答】Results:The MTT experiment showed that overexpression of the SNTB1 gene can enhance the viability of HCT116 and LOVO cells (P<0.01). Compared with the HCT116/NC and LOVO/NC groups, the viability of cells in the HCT-116/OE and LOVO/OE groups was significantly increased. The colony formation experiment showed that overexpression of the SNTB1 gene can significantly promote the sphere-forming ability and proliferation of colon cancer cells. Compared with the HCT-116/NC and LOVO/NC groups, the cloning ability of cells in the HCT-116/OE and LOVO/OE groups was significantly improved, with significant differences (P<0.01). The Transwell and Invasion Scratch experiments showed that the migration, invasion, and scratch healing rate of HCT-116/OE and LOVO/OE colon cancer cells were higher than those of the HCT-116/NC and LOVO/NC groups (P<0.01), indicating that overexpression of the SNTB1 gene can promote the metastasis of colon cancer cells. qPCR detected the expression of SNTB1 in each group【回答】结果:1MTT实验表明SNTB1基因过表达能增强HCT116及LOVO细胞的活力(P<0.01)，与HCT116/NC组和LOVO/NC组相比，HCT-116/OE组与LOVO/OE组细胞的活力显著增加;集落形成实验表明SNTB1基因过表达能显著促进肠癌细胞成球能力与增殖情况，与HC-116/NC组和LOVO/NC组与LOVO/OE组细胞的克隆球形成能力显著提高，结果具有显著性差异(P<0.01)。Transwell、InvasionScratch实验表明转染后的HCT-116/OE组LOVO/OE组肠癌细胞其迁移侵袭及划痕应合率都高于HCT-116/NC组和LOVO/NC组(P<0.01)，其结果表明过表达SNTB1基因可促进大肠癌细胞的转移。qPCR法检测转染后各组SNTB1结果显示，与HCT-116/NC组和LOVO/NC组相比，HCT116/OE组及LOVO/OE组肠癌细胞的SNTB1的mRNA表达量显著增加(P<0.01)。Western blot结果显示，与HCT116/NC组和LOVO/NC组相比，HCT116/OE组及LOVO/OE组肠癌细胞的SNTB1的蛋白表达量及pMAPK12/MAPK12的比值显著升高(P<0.01)。2.经过八宝丹干预后，MTT实验结果显示，与HCT-116/NC组及LOVO/NC组的细胞相比，HCT116/SNTB1组及LOVO/SNTB1组的肠癌细胞的增殖能力显著提高(P<0.01)HCT-116/NC+BBD组与LOVO/NC+BBD组的细胞增殖水平降低(P<0.01)。八宝丹干预过后，HCT-116/SNTB1+BBD组及LOVO/SNTB1+BBD组的肠癌细胞增殖能力有所下降(P<0.05)这些结果表明八宝丹能通过影响SNTB1基因表达水平，降低肠癌细胞的增殖情况。这段的【回答】The Transwell, Invasion, and Scratch experiments showed that after intervention with BaBaoDan, the migration, invasion, and scratch healing rate of HCT116/NC and LOVO/NC colon cancer cells were significantly decreased (P<0.01), indicating that BaBaoDan can inhibit the metastasis of colon cancer cells. Additionally, compared with HCT116/SNTB1 and LOVO/SNTB1 overexpressing cells, the migration, invasion, and scratch healing rate of HCT116/SNTB1+BBD and LOVO/SNTB1+BBD colon cancer cells were also decreased (P<0.05). These results suggest that BaBaoDan can reduce the metastasis of colon cancer cells by affecting the expression level of the SNTB1 gene.In the qPCR experiment, it was found that the mRNA expression levels of SNTB1, MAPK12, and p-MAPK12 in HCT-116/NC+BBD and LOVO/NC+BBD groups were significantly decreased compared with the HCT-116/NC and LOVO/NC groups (P<0.05). Similarly, the mRNA expression levels of SNTB1 and MAPK12, as well as the protein expression level of SNTB1 and the【回答】TanswellInvasion、Scratch实验表明在HCT116/NCLOVO/NC组细胞中加入八宝丹的干预后，其迁移侵袭及划痕愈合率都较NC组细胞有明显的下降(P<0.01)其结果表明使用八宝丹可抑制大肠癌细胞的转移:同时，与HCT116/SNTB1及LOVO/SNTB1过表达细胞株相比，八宝丹使用后的HCT116/SNTB1+BBD与LOVO/SNTB1+BBD组肠癌细胞的迁移侵袭及划痕愈合率都有所下降(P<0.05)。这些结果表明八宝丹能通过影响SNTB1基因表达水平，降低肠癌细胞的转移在qPCR实验中，我们发现与HCT116/NCLOVO/NC组相比，HCT-116/NC+BBDLOVO/NC+BBD组的SNTB1、MAPK12、P-MAPK12Ln&rLOVO/SNTB1组相比，HCT116/SNTB1+BBD与LOVO/SNTB1+BBD组细胞中的SNTB1、MAPK12p-MAPK12mRNA表达水平下降，表明八宝丹可降低大肠癌细胞中的SNTB1、MAPK12、P-MAPK12等基因的mRNA表达水平(P<0.05)。Westemn blot结果显示，与HCT116/NC、LOVONC组相比，HCT116/NC+BBD、LOVO/NC+BBD的SNTB1蛋白表达量、P-MAPK12/MAPK12的比值下降(P<0.01);与HCT-116/SNTB1及LOVO/SNTB1组相比，HCT116/SNTB1+BBD与LOVO/SNTB1+BBD组细胞中的SNTB1蛋白表达量、P-MAPK12mRNA表达水平下降，表明八宝丹可能是通过介导SNTB1，进而发挥调控p38MAPK信号通路，抑制大肠癌发生发展的作用。这段的【回答】Compared with the Control group, the Model group of mice showed a decline in their living conditions (including fur luster, activity, and diet), and the Model/OE group with SNTB1 overexpression exhibited significant weight loss, bloody stool, and diarrhea (P<0.001). However, the severity of the disease in the Model/Sh group with SNTB1 knockdown was not significant, indicating that SNTB1 may be a potential target gene that promotes the development of colitis-associated colorectal cancer. The small animal live imaging results showed that after injection of adenovirus for 2-3 weeks, the colon tissues of the Model/OE-NC group, Model/OE group, Model/Sh-NC group, and Model/Sh group of mice showed strong fluorescence under the small animal live imaging system, indicating that the adenovirus had infected the colon tissues. The IHC staining results showed that compared with the Model group, the positive expression levels of SNTB1, MAPK12, and p-MAPK12 in the colon tissues of the Model/OE group【回答】3与Control组相比，Model组小鼠生活状态(皮毛光泽、活动状态、饮食等)下降，SNTB1过表达的Model/OE组小鼠出现明显的体重下降便血及腹泻(P<001)，而SNTB1敲减的Model/Sh组小鼠疾病严重程度则不明显，表明SNTB1可能是促进肠炎相关性肠癌发展过程中的潜在靶基因。小动物活体成像结果显示，在注射腺病毒2~3周后，Model/OE-NC组、Model/OE组、Model/Sh-NC组、Model/Sh组小鼠的结肠组织在小动物活体成像系统下观察可呈强荧光，说明腺病毒已感染了结肠组织。IHC染色结果发现，与Model组相比，Model/OE组结肠组织中SNTB1、MAPK12PMAPK12的阳性表达量明显升高(P<0.01)，而Model/Sh组SNTB1、MAPK12PMAPK12的阳性表达量降低。qPCR实验结果显示与Model组相比，Model/OE组SNTB1的mRNA表达量显著增高(P<0.05)，而Model/Sh中的SNTB1mRNA表达量显著降低(P<0.05)。Western blot结果显示，与Model组相比，SNTB1过表达组的SNTB1蛋白表达量、P-MAPK12/MAPK12的比值升高(P<0.01)而SNTB1敲减组的SNTB1蛋白表达量、PMAPK12/MAPK12的比值显著降低(P<001)。这段的【回答】The results of the study showed that Ba Bao Dan could inhibit the proliferation and metastasis of colorectal cancer cells by regulating the expression of SNTB1 and the p38MAPK signaling pathway in the cells, as well as relieve the disease activity index, improve the survival status and pathological condition of mice. SNTB1 may be an important target for Ba Bao Dan to inhibit the development of colitis-associated colorectal cancer. Keywords include Ba Bao Dan, SNTB1, p38MAPK12 signaling pathway, colitis-associated colorectal cancer, metastasis.After intervention with Ba Bao Dan, the results of the sample showed that compared with the Model+AAV-NC group, the disease activity of mice in the Model/NC+BBD group and the Model/OE-SNTB1+BBD group improved, with a decrease in severity, weight loss, and relief of rectal bleeding and diarrhea index (P<0.05). IHC staining results showed that compared with the Model+AAV-NC group, the number of SNTB1 MAPK12 and P-MAPK12 positive cells in the Model+【回答】4.加入八宝丹干预后，取材结果显示，与Model+AAV-NC组相比，Model/NC+BBD组及Model/OE-SNTB1+BBD组小鼠疾病活动情况有所改善，严重程度降低，体重下降、便血及腹泻指数有所缓解(P<0.05)。IHC染色结果发现，与Model+AAV-NC组相比，Model+AAV/NC+BBD组SNTB1MAPK12、P-MAPK12阳性细胞数量显著降低(P<001)，而Model/OE-SNTB1+BBD(P<005)组的SNTB1、MAPK12、p-MAPK12阳性率也有所下降(P<0.05)。HE染色结果显示，与Model+AAV-NC组相比，Model+AAV/NC+BBD组的小鼠结肠组织炎症细胞浸润的情况有所缓解，肠道组织结构更为完整(P<0.05)。qPCR实验结果显示，与Model+AAV-NC组相比，Model+AAV/NC+BBD组SNTB1的mRNA表达量显著下降(P<0.01);Model/OE-SNTB1+BBD组SNTB1的mRNA表达量有所下调(P<005)。Westernblot结果显示，与Model+AAV-NC组相比MAPK12/MAPK12的比值显著降低(P<0.01)而Model/OE-SNTB1+BBD组SNTB1蛋白表达量、pMAPK12/MAPK12的比值有所下降(P<005)。结论:SNTB1可能是八宝丹抑制结肠炎相关性结肠癌生成的重要靶标，过表达SNTB1能增强肠癌细胞的活力、增殖及转移能力，加重结肠炎相关性结肠癌小鼠的疾病严重程度。而使用八宝丹干预后，八宝丹可通过介导大肠癌细胞中SNTB1的表达，调控p38MAPK信号通路，进而发挥抑制大肠癌细胞增殖及转移的作用。同时八宝丹也可以靶向调节SNTB1的表达，缓解疾病活动指数，改善小鼠生存状态及病理情况;八宝丹也可以调控p38MAPK信号通路，从而发挥其抑制大肠癌转移和治疗大肠癌的目的。关键词:八宝丹，SNTB1，p38MAPK12信号通路，结肠炎相关性结肠癌，转移这段的【回答】亲 完结了哦【回答】
